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It works by activating the same receptors as GLP-1, helping to reduce hunger and increase feelings of fullness

As an incretin hormone, GLP-1 enhances insulin secretion, suppresses glucagon release, delays gastric emptying, and modulates appetite regulation

Together, these findings suggest that CBS refuels cysteine pools for GSH synthesis through de novo cysteine biosynthesis under cystine restricted conditions

The GLP-1 drugs studied included liraglutide, semaglutide, dulaglutide, exenatide, lixisenatide and albiglutide

These drugs help regulate blood sugar by triggering insulin release and suppressing glucagon

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